June 2025
Dr. John Hill
Scott Wildenheim
Caleb Ferroni
Ray Pace
🎧 Episode Summary:
In this engaging episode of CardioCast, Scott, Caleb, Ray, and Dr. John Hill take a deep dive into the clinical nuances of narrow complex rhythms—including both tachycardias and bradycardias. More than a rhythm review, this episode emphasizes the critical thinking needed to understand underlying causes, interpret subtle EKG findings, and choose appropriate interventions—whether pharmacologic or electrical. From sinus tach to SVT, AFib with RVR, WPW, and bradycardia management, this is a must-listen for any EMS provider aiming to master rhythm interpretation and prehospital cardiology care.
🧭 Key Topics Covered:
Narrow QRS defined as ≤ 0.12 seconds (120 ms)
Importance of thinking in milliseconds (ms) over decimal seconds for accuracy
EMS versus medical education perspectives on QRS measurement
Rate > 100 = tachycardia; < 60 = bradycardia
Use of the computer’s timing data for accurate interval interpretation
Visual assessment still crucial—trust, but verify
Not all tachycardia is pathological—evaluate for cause (e.g., dehydration, fever, exertion)
Always correlate rhythm with history, exam, and vitals
Common mistake: ignoring compensatory tachycardia in conditions like sepsis
Controlled vs. uncontrolled AFib
Clinical considerations before treating rate: Is the AFib the primary issue or compensatory?
When to initiate treatment and when to observe
Target rate ranges and ED discharge criteria
Dangers of oversimplifying SVT as “HR > 150”
Differentiating SVT from Flutter with 2:1 block (~150 bpm)
Role of vagal maneuvers and adenosine—why diagnostic use is risky
Emphasis on morphology and P-wave analysis
Recognizing delta waves and shortened PR intervals
Why adenosine may be dangerous in WPW
Safe approach: defer to ED or cardiology when WPW is suspected
Most patients tolerate slow rates well if perfusion is intact
First-line: Assess for parasympathetic stimulation (consider atropine if present)
Push-dose epi for augmenting sympathetic tone—preferred in structural or medication-induced bradycardias
Fluid bolus often underutilized and low-risk
Indications for transcutaneous pacing in unstable bradycardia
Recommended initial settings (e.g., rate 60 bpm, start at 20 mA)
Tips for verifying electrical AND mechanical capture
Utilizing the "pause" feature to document underlying rhythm
Cardioversion is often safer than pharmacologic interventions
Always reassess sync settings between shocks
Don’t over-rely on adenosine—requires full preparedness for adverse effects
Sedation optional for urgent cardioversion (atomidate preferred)
Rate vs. Rhythm control: Beta blockers, calcium channel blockers, amiodarone, digoxin
Red box protocols for beta blockers to avoid harm
Push-dose epi: evolving dosing practice (10–50 mcg as needed)
Amiodarone as a fourth-line agent in AFib with RVR
Role of fluids as diagnostic and therapeutic tool across both tachy- and bradyarrhythmias
Over-reliance on technology vs. touching the patient
Misinterpretation of devices (e.g., pleth mismatch with monitor tracing)
4-lead vs. 12-lead utilization strategy
Equipment training gaps (e.g., “Pause” button, shock dumping, monitor syncing)
💡 Clinical Pearls:
Always ask why the heart rate is abnormal—tachycardia and bradycardia are symptoms, not diagnoses.
Know your monitor inside and out—many don’t realize what half the buttons do.
When in doubt, electricity is often safer than medication for rhythm correction.
Fluid is a therapeutic trial—response can guide treatment.
Touch the patient. Monitors lie. Patients don’t.
Caleb discusses sinus tachycardia
John reminds viewers to ask "why"?
Ray describes proper pad placement for cardioversion
Scott explains what makes the SVT digagnosis